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In line with this, a ROS/RNS-dependent regulation of IL-6 was envisaged in response to muscle exercise as shown by an impaired release of this cytokine after administration of vitamins A, C and E or inactivation of NOS by N()-nitro-l-arginine methyl ester (L-NAME) [110,111]

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(1996) rats were fed a diet of 100 IU (67 mg) of vitamin E/kg of feed

how mitochondrial-derived signals (e.g., specific lipids, peptides, or ROS subsets) propagate to regulate cytoplasmic iron metabolism, lipid peroxidation, or antioxidant systems remains elusive