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Following incubation with 10 U Dpn I (New England Biolabs, Frankfurt, Germany) to eliminate the remaining vector DNA, reactions were used to transform E

The pathophysiology of doxorubicin and ischemia/reperfusion-induced myocardial injury in mice may be primarily associated with increased mitochondrial iron levels and lipid peroxidation, suggesting that mitochondrial oxidative damage is a principal mechanism underlying ferroptosis in this context [107]

Besides the aforementioned pipelines, other bioinformatic approaches have been developed in recent years for the identification of DMRs using array data [103,104,105,106,107]